Objective: Intervertebral disc degeneration is associated with decreased proteoglycan ( PG ) synthesis by disc chondrocyte like cells.
目的: 椎间盘退变伴随着蛋白 多糖 的减少,骨形态发生蛋白有促进蛋白多糖生成的作用.
Conclusion: The synthesis of PG in chondrocyte in mandibular condyle may be affected by abnormal stress.
结论: 异常负荷可导致软骨细胞蛋白多糖合成的减少及其构成的变化,从而使髁突纤维软骨的粘弹性降低.
Conclusion: Apoptosis of chondrocyte plays an important role in the pathomechanism of OA.
结论: 软骨细胞凋亡在骨关节炎发病中具有重要的作用.